Previous report showed that SLC16A3 might be involved in the tissue-specific transplantation antigen P35B (TSTA3)-activated network mediated immune response in hepatitis/cirrhotic tissues (HBV or HCV infection), such as T-cell homeostasis, neutrophil chemotaxis, interleukin-8 expression, immune response, and B-cell activation [26, 27]. This evidence concerns the gene SLC16A3 and hepatitis A virus infection.