Genetic analyses of known driver genes (CTNNB1, TP53, CDKN2A, RB1 and MEN1) and of genes recently reported to be of importance in ACC (ZNRF3, DAXX, TERT and MED12) revealed in the original patient tumor (also representing passage 1) a somatic mutation in TP53 (a frameshift deletion: Hg19 positions: 7574003 on Chr 17: G is deleted) while the tumor was devoid of mutations in any of the other investigated genes (T91/L91 in [1]). Here, CDKN2A is linked to neoplasm.