DM1 myotubes transduced with KCa1.1-BacMam showed a 3.5-fold increase in myogenin levels compared with untreated DM1 myotubes, the latter displaying only 1.5-fold increase in myogenin expression from day 0 myoblasts (Figure 7h). This evidence concerns the gene KCNMA1 and myotonic dystrophy type 1.