KCNMA1 and myotonic dystrophy type 1: 16, 50, 52 Although the current study focuses on the consequences of the loss of plasma membrane expression of KCa1.1 in DM1 myoblasts, future work aimed at identifying the splice variants of α-, β-, and γ-subunits of KCa1.1 in normal and DM1 myoblasts is needed to understand the causes of this loss.