CCNB1 and pancreatic ductal adenocarcinoma: Concordantly with our results, Li et al. reported that RACK1 was highly expressed in pancreatic ductal adenocarcinoma (PDAC) and could induce G1/S cell cycle arrest by decreasing the expression of Cyclin D1 [31], while Zhang et al. reported that overexpression of RACK1 contributed to the progression of oral squamous cell carcinoma (OSCC) and stable silencing of RACK1 resulted in a distinct G1 and G2 phase arrest by downregulating Cyclin B1 and Cyclin D1 [33].