The origin of altered glycosylation in cancer has been investigated by Haab’s group [73] Specifically, to test the hypothesis that pro-inflammatory stimuli affect mucin glycosylation, they stimulated six pancreatic cancer cell lines with IFN-γ, interleukin-1 (IL-1), and TNF-α and evaluated alterations in glycosylation and expression. Here, MUC5AC is linked to cancer.