As it is possible that PUFAs in EGFR mutant cells might play a role in sustaining the higher EGFR activation rate, the mechanistic function of PUFAs in EGFR and non-EGFR driven NSCLC should be further elucidated to determine its effect on the growth inhibition or promotion of EGFR mutant tumours64, 68. This evidence concerns the gene EGFR and non-small cell lung carcinoma.