Since we observed the increase in the immature CD11b− cNK cell subsets and, simultaneously, an increase in highly mature KLRG1+ cNK cells after RH and ME49 infections, we, next, assessed the distribution of KLRG1+ cNK cells between CD11b− (R0, R1) and CD11b+ (R2, R3) subsets. The gene discussed is KLRG1; the disease is infection.