Our initial experiments showed that replenishing ECG in glucose- and glutamine-deprived OCI-LY10 DLBCL cells stimulated cell viability (Figure 5A) and induced both NF-κB-p65 and NFATc1 activation (Figure 5B), suggesting that ECG can incorporate into the HBP through GlcNAc protein modification. Here, NFATC1 is linked to diffuse large B-cell lymphoma.