APOA1 and inflammation: Similarly, administration of apoA-I mimetic peptides, human apoA-I, or over-expression of human apoA-I via a recombinant adenoviral vector, protected rodents from developing neutrophilic airway inflammation and ALI in models of LPS- or LTA-mediated systemic inflammation (Yan et al., 2006; Jiao and Wu, 2008; Li et al., 2008; Van Linthout et al., 2011; Kwon et al., 2012; Sharifov et al., 2013).