Chen et al. reported that posttreatment with magnolol, an active ingredient of Magnolia officinalis (Hou Pu), ameliorates cerebral infarction partially by dose-dependently inhibiting iNOS, TNF-α, IL-1β, and IL-6 expression in the ischemic area 24 h after transient global ischemia [50]. The gene discussed is IL1B; the disease is brain infarction.