Even at this chronic stage, IFN-γ and TNF persist as essential cytokines, maintaining the migration of T cells to consolidate the cardiomyopathy by increasing the expression of CCL5 (RANTES), CCL2 (MCP-1), CXCL10 (IP-10), and CXCL9 (MIG) as well as enhancing intercellular adhesion and vascular cell adhesion molecules [7, 12]. The gene discussed is CXCL9; the disease is cardiomyopathy.