An increase in amino acid transport may be expected in cancer, most likely as the result of increased amino acid demand for energy, protein synthesis and cell division: surprisingly, S5 Fig shows that SLC-encoding genes down-regulated in NIH-RAS compared to NIH3T3 cells are enriched in genes encoding amino acid transport, particularly of neutral amino acids (e.g. the SBAT1-encoding Slc6a15 gene). This evidence concerns the gene SLC6A15 and cancer.