Though the role of inflammation in IPF, specially from Th2 background, including the treatment of IPF with anti-inflammatories is hotly debated [6], the expression of Th2 cytokines, specifically IL-4 and IL-5, predominate over Th1 cytokine, IFN-gamma, as accredited to play an important role for progression of pulmonary fibrosis [7]. Here, IL5 is linked to idiopathic pulmonary fibrosis.