The PSGs in T. asiatica are more concentrated to gene families involved in internal homeostasis (for example, carbonic anhydrase), tegumental development (for example, cytoskeletal proteins, cell junction proteins and glycosyltransferases) and lipid uptake (for example, LDLR and fatty acid-binding protein), which are probably associated with the establishment of new immune-evasion and nutrient uptake strategies at the lipid-rich infection site (liver) in a new intermediate host (pigs). The gene discussed is LDLR; the disease is infection.