Specifically, the results showed (i) decreased levels of αβ‐crystallin, a chaperone for VEGF‐A, and (ii) increased levels of LTBP1 and LTBP2, regulators of TGF‐β availability 28, as well as of OSTP and FMOD, which are components of the extracellular matrix and might be involved in the paracrine action of T2DM‐BMMSCs on endothelial cells 29, 30, 31, 32. The gene discussed is TGFB1; the disease is type 2 diabetes mellitus.