ANXA1 and Huntington disease: Although dysregulation of ANXA1, a putative target of miR-196a, in HD has strongly suggested the role of miR-196a in regulating neural cell response to stress and pathogenic changes in cells such as the accumulation of mHTT aggregates, continued effort in identifying gene targets of miR-196a that were dynamically changed during HD progression will lead to insight on the role of miR-196a in HD pathogenesis.