Thus, we conducted this study with the aim to evaluate 1) the association between EAT, the other metabolic parameters and the severity of steatosis 2) the relationship between cardiovascular (cIMT, cplaques, E/A), liver (presence of NASH and significant fibrosis) damage and metabolic risk factors including EAT and 3) the relationship between EAT and genetic factors strongly influencing liver steatosis, including PNPLA3 and TMS6SF2. The gene discussed is PNPLA3; the disease is metabolic dysfunction-associated steatohepatitis.