In addition, our experiments using phosphorylation site-specific antibodies showed that ATP-induced synaptic depression from hippocampal slices10 is associated with a fast and reversible dephosphorylation of GluA1 at the CaMKII phosphorylation site S567 without change in the phosphorylation levels of S845 or S831. This evidence concerns the gene GRIA1 and depressive symptom measurement.