We have recently demonstrated that the S. aureus‐CFS used in our study, potently induces FOXP3+ cells and promotes a diverse phenotype of these cells with production of both regulatory (IL10) and pro‐inflammatory (IFNγ and IL17) cytokines in a partly monocyte‐dependent manner 43. The gene discussed is FOXP3; the disease is myalgic encephalomeyelitis/chronic fatigue syndrome.