Among several proteins whose malfunction has been linked to molecular events leading to neuronal dysfunction in Parkinson's disease (PD), α-synuclein deservingly hold the status of a primary culprit due to its causative role in certain familial forms and a pivotal role in the formation of histopathological hallmarks in both familial and idiopathic forms of the disease [reviewed in (Dev et al., 2003, Venda et al., 2010)]. This evidence concerns the gene SNCA and Parkinson disease.