We also checked the response of S6-ribosomal protein (rpS6), which is downstream Akt; of GSK3α/β, which are phosphorylated by Akt and ERK [40, 41]; and of p38-MAPK, also described as a target of quercetin or quercetin-derived analogs in leukemia cells [42, 43]. This evidence concerns the gene AKT1 and leukemia.