Of note, however, Undas et al. reported decreased clot permeability and longer clot lysis time in both healthy subjects and patients with myocardial infarction that have increased Lp(a) levels [a cutoff value of Lp(a) 300 μg/ml] as a result of small apo(a) size isoforms (Undas et al. 2006). This evidence concerns the gene LPA and myocardial infarction.