Although the mechanisms by which the cancer cells evade anoikis are unknown, the gain of miR-200c expression in endometrial and breast cancer in cultures restored the sensitivity of these cells toward anoikis, through suppression of neurotrophic-tyrosine-receptor kinase type 2, NTRK2 (Howe et al., 2011). The gene discussed is NTRK2; the disease is breast carcinoma.