In a study on ovarian cancer, miR-200c expression led to suppression of Fas-associated phosphatase-1 (FAP1) and increased CD95, a death receptor, surface expression and consequently led to increased cell sensitivity to CD95-mediated apoptosis during CD95 agonist treatment (Schickel et al., 2010), therefore restoring the tumor cells sensitivity toward CD95-mediated apoptosis. This evidence concerns the gene FAS and neoplasm.