In C57BL/6 mice at 1 week post-stroke, despite substantial resolution, G-CSF, GM-CSF, IFNγ, IL-1α, IL-1β, IL-4, IL-5, IL-6, IL-10, IL-12 (p40), IL-12 (p70), IP-10, KC, MCP-1, MIP-1α, MIP-1β, MIP-2A, RANTES, and TNFα remained elevated in the infarct compared to the equivalent area of cortex from sham mice (Fig. 3b, left graph). This evidence concerns the gene CCL5 and stroke disorder.