However, the over-expression of HSP-70 in the cerebral cortex of animals exposed to 900 or 2450 MHz RF attenuated caspase-3 [10] and inhibited neuronal apoptosis [40], thereby diminishing cerebral ischemia in the astrocytes [41] and maintaining the mitochondrial physiology during induced stress through glucose deprivation in the astrocytes [42]. Here, CASP3 is linked to brain ischemia.