PDILT and chronic kidney disease: Smaller credible sets, in terms of the number of SNPs they contain, or the genomic interval that they cover, thus correspond to more precise fine-mapping. The 99% credible set at the PDILT-UMOD locus included a single variant (rs77924615, πC>0.999), which maps to an intron of PDILT. This variant has previously been reported as driving the primary association signal for CKD at the PDILT-UMOD locus through whole-genome sequencing and long-range haplotype imputation into 194,286 Icelandic individuals with serum creatinine measurements.34