More recently, Bonneman et al. [2] reported on the currently recognized of CMD entities: a) laminin alpha 2 related CMD (primary laminin 2/merosin deficiency); b) alpha-dystroglycan related dystrophies; c) congenital disorders of glycosylation (CDG) with abnormal alpha-dystroglycan glycosylation; d) collagen VI and Integrin-related CMD forms; e) Intracellular and nuclear CMD forms. Here, LAMA2 is linked to congenital disorder of glycosylation.