The main autoimmune etiology of MS consists of activated IFN-γ-producing T helper 1 (Th1) cells, that recognize peptides of the myelin sheath, including myelin basic protein (MBP), proteolipid protein (PLP), and myelin oligodendrocyte glycoprotein (MOG) [3], as well as IL-17-producing Th17 cells, a subset of CD4+ T-cells shown to be involved in the pathogenesis of autoimmune diseases [6]. This evidence concerns the gene MOG and myeloid sarcoma.