As our aforementioned findings (Figure 6) indicate that WNT4 is downstream of the BRCA1/NEAT1/miR-129-5p axis and activates oncogenic WNT signaling in breast tumor cells, we postulated that WNT4 is involved in enhancing malignant phenotypes and stemness of BRCA1-deficient breast tumor cells. Here, NEAT1 is linked to breast neoplasm.