The fact that CXCL8 increased migration of both TPC-1 and BCPAP cells together with the fact that treatment with IFNγ produced a reduction in the number of migrated cells only in BCPAP (in which IFNγ significantly lowered the secretion of CXCL8) constitutes a further confirmation of the tumor-promoting effect of CXCL8 and of the need to find novel and more effective inhibitory strategies. Here, IFNG is linked to neoplasm.