EPHA2 and non-small cell lung carcinoma: As we previously found p38MAPK to be in complex with IGF-1R and to be a pro-survival factor in NSCLC cells [30] our results may also indicate that crosstalk exists between EphA2 and IGF-1R similarly as reported for EphA2/EGFR [27] allowing certain EphA2 signaling and proliferation capacity to be maintained.