We detected markedly increased levels of the corresponding chemokines CCL2 (CCR2 ligand), CCL3 and CCL4 (CCR5 ligands), and CCL20 (CCR6 ligand) in early peritonitis (Fig. 1E), demonstrating a substantial production of inflammatory chemokines with the potential to attract unconventional T cells to the site of infection, thereby complementing the local pool of tissue-resident Vγ9/Vδ2 T cells and MAIT cells. This evidence concerns the gene CCL3 and peritonitis.