Although many mutations are considered breed specific [3], others such as the ones underlying degenerative myelopathy (DM; [9]), hyperuricosuria (HUU; [10]) or factor VII (FVII) deficiency [11,12] are thought to be ancient in their origins and therefore potentially widespread across breeds. This evidence concerns the gene F7 and hyperinsulinemic hypoglycemia, familial, 4.