We measured inflammatory cytokines in the bronchoalveolar lavage (BAL) fluid throughout IAV infection and found that IFNα treatment significantly increased secretion of IL‐6, IP‐10, MCP‐1 and other proinflammatory cytokines from day 4 post‐infection onwards, while the cytokine response in the IFNλ treatment group was comparable to infected, vehicle‐treated control mice (Fig 2A). The gene discussed is IFNA1; the disease is infection.