DCDC2 and dyslexia: This hypothesis is supported by previous studies on mice mutated in other candidate dyslexia susceptibility homologous genes, such as Dcdc2 and Dyx1c1. These animals do not show the expected migration deficits, and lack lamination defects or clear anomalies in the cortex; however, changes in learning, memory or auditory processing have been reported in both cases (Gabel et al. 2011; Truong et al. 2014; Rendall et al. 2015).