We found twenty-four out of seventy-one exon features in GAK had FDR-significant increased expression at FDR-level of significance in PD relative to control (Fig 1A); additionally, none of the exon features in GAK showed significantly decreased expression in PD, and those that are increased in PD are exclusively located in the 3’ region of the gene (Fig 1A). Here, GAK is linked to Parkinson disease.