Given that mutations in B3GNT1 lead to WWS and that it is classified as a dystroglycanopathy [14,15], a group of muscular disorders whose etiology is hypothesised to be caused by aberrant glycosylation of dystroglycan [35], we tested whether or not the C. elegans dystroglycan homologs, dgn-1, dgn-2 and dgn-3, exhibited dye-filling phenotypes. The gene discussed is B3GNT2; the disease is neuromuscular disease caused by qualitative or quantitative defects of alpha-dystroglycan.