For example, FLI1 repositions in 100% (10/10) of breast cancers and 92.9% (13/14) of prostate cancers (Leshner et al., 2016; Meaburn K. J. et al., 2016), rates far too high to suggest it can be used to stratify indolent and aggressive cancers. This evidence concerns the gene FLI1 and prostate carcinoma.