In a GWAS focusing on occupational exposures, CTNNA3 (catenin alpha 3, alpha-T catenin) was reported to be the strongest candidate gene for toluene diisocyanate (TDI)-induced asthma in Korean patients (Kim et al. 2009), and only one GEWIS has been published that identified novel susceptibility loci for occupational exposure to biological dust, mineral dust, and gases and fumes in relation to forced expiratory volume in 1 sec (FEV1) levels (de Jong et al. 2015). The gene discussed is CTNNA3; the disease is asthma.