Regarding SNP polymorphisms, the obtained results showed the lack of the GSTA1 C69T polymorphism effect on cRCC risk, since the carriers of GSTA1 CT+TT (low-activity) genotype were not at increased risk of cRCC in comparison with individuals with GSTA1-active genotype (OR = 1.19, 95%CI:0.63–2.25, p = 0.58). This evidence concerns the gene GSTA1 and chromophobe renal cell carcinoma.