Therefore, even if the coefficients of correlation were relatively low, based on the significance obtained by two different statistical tests (Mann-Whiney and Pearson's exact tests), we could assume that in a group of DLBCLs, the upregulation of BCL6 may be due to the downregulation of PATZ1, whichcould also be directly responsible for the downregulation of BAX in the same group of lymphomas, likely resulting in an anti-apoptotic effect. Here, PATZ1 is linked to lymphoma.