FGF2 and endometrial cancer: Since [NSIS6S]-[NSIS]5 was the most potent inhibitor of FGF2-dependent HUVEC proliferation, migration and sprouting (Fig 2), we investigated the degree to which [NSIS6S]-[NSIS]5 reduces endothelial tube formation induced by conditioned medium generated by endometrial cancer cells expressing exogenous secreted form of FGF2 (FGF2-B9).