A more recent study reported that lncRNA NUTF2P3-001 function as ceRNAs to communicate with KRAS mRNA by competitively binding to miR-3923, and the overexpression of NUTF2P3-001 will unregulated KRAS expression by depriving the inhibition of miR-3923 on KRAS leading to the proliferation and invasion of pancreatic cancer cell [19], revealing the functional roles of lncRNA-mediated ceRNA crosstalk in PC for the first time. This evidence concerns the gene KRAS and pancreatic neoplasm.