MET and non-small cell lung carcinoma: Recent molecular characterization of patient samples demonstrates that NSCLC arises from alteration of a relatively small subset of genes [4–6], including MET copy number (CN) gain and exon 14 skipping, which together account for ~6.5% and 3.6% of driver mutations in lung adenocarcinoma (LUAD) and lung squamous cell carcinoma (LUSC) cases, respectively [4, 7].