As we showed that lung cancer cell lines sensitive to EGFR inhibitors expressed a trend towards higher baseline levels of MHC-I and PD-L1, and also higher expression of PD-L1, MHC-I, and MHC-II in response to IFNγ, we investigated whether PD-L1 overexpression is associated with overexpression of other immune-related genes in 186 lung cancer cell lines from CCLE database. Here, EGFR is linked to lung carcinoma.