DTNBP1 and schizophrenia: Moreover, sandy mice, which carry a dysbindin-1 null mutation (dysbindin-1−/−, Sdy), display electrophysiological deficits in auditory evoked response adaptation, prepulse inhibition and evoked γ-activity, similar to those electroencephalogram (EEG) patterns in patients with schizophrenia and schizophrenia-like behaviors such as locomotion and cognitional deficits [20–22].