ABCA1 and infection: Unlike our data which supports upregulation of ABCA1 following mycobacterial infection (Figures 1–4), infection of lung epithelial cells with another intracellular pathogen, Chlamydia pneumoniae specifically downregulated ABCA1 (Korhonen et al., 2013) via upregulation of miR-33 (Zhao et al., 2014) and this was associated with decreased chlamydial survival within the host cells (Korhonen et al., 2013).