Pre-miR-29a treatment in diabetic mice significantly restored the levels of β-catenin (Fig. 2c), and prevented upregulation of profibrotic factors, including TGF-β1 (Fig. 2d), fibronectin (Fig. 2e), and DKK1 (Fig. 2f), induced by hyperglycemia. Here, FN1 is linked to Hyperglycemia.