Our recent report also supports the critical role of GSH and redox regulation in the myelination processes and white matter maturation.37, 38 At the cellular level, research in the same model showed that hippocampus fast-spiking parvalbumin γ-aminobutyric acid interneurons and their synchronization are also impaired, all features known to be affected in schizophrenia.39 In summary, preclinical research indicates that redox imbalance affects the fornix and hippocampus, with relevance to schizophrenia. The gene discussed is PVALB; the disease is schizophrenia.