Despite these interesting results and the regulatory data observed for all these SNPs, the lack of information regarding T2D status among MM patients did not allow us to ensure that the observed effect of the TCF7L2, SLC2A2, PRC1, and PROX1 SNPs on OS could not be due to a different distribution of diabetic patients when grouping by genotype or gender. This evidence concerns the gene PRC1 and Miyoshi myopathy.